Seborrheic dermatitis, the dermatitis of the sebaceous areas, is a common disease. It may be considered part of a clinical spectrum which include dandruff and psoriasis
Seborrheic dermatitis is a common skin disease associated with increased and altered sebum production and yeast colonization. Seborrheic dermatitis presents as a chronic dermatitis or eczema in sebum rich areas of the body like scalp, face and upper trunk. Its milder, non-inflammatory form is known as dandruff.
Seborrheic Dermatitis affects 1-3% general population, though dandruff is a very common occurrence. More than 36% of HIV positive patients have Seborrheic Dermatitis.
What are the Causes for Seborrheic Dermatitis?
Seborrheic dermatitis appears in genetically pre-disposed individuals with the so called seborrheic diathesis, meaning increased sebum secretion in the sebaceous gland rich areas of the body- the scalp, face, front of chest, and in between the shoulder blades. Certain triggering agents make the greasiness of the skin in these areas a flourishing ground for the yeasts known as malassezia. These yeasts breakdown and alter the composition of the sebum which further sets in motion an inflammatory response in the skin. This in turn increases the sebum secretion and the cycle continues again.
The skin becomes red, irritated and scaly. Itching is prominent in acute, active state. In addition to the classical sites, seborrheic dermatitis can also affect the eyelids, armpits, groin and gluteal fold.
Malassezia species M.restricta and M.globosa have been isolated from the seborrheic dermatitis skin.
Seborrheic dermatitis is seen in the first few months of life when maternal androgens are present in the blood. Then, the disease re appears in susceptible individuals after the sebaceous glands become active during puberty.
Seborrheic dermatitis can be considered a part of the clinical spectrum between dandruff and psoriasis.
What are the Triggers for Seborrheic Dermatitis?
Stress & Fatigue. Both lowered immunity and hormonal stimulation are the reasons for increased seborrheic dermatitis in stressed and fatigued conditions.
Hormonal Triggers . Androgen hormones control the sebaceous activity in humans. Probably these are under the control of a sebotrophic hormone secreted from the hypothalamus. Body builders using anabolic steroids get severe attacks of seborrheic dermatitis.
Environmental Triggers. Seborrheic dermatitis is precipitated in low humid conditions and in winter.
Low Immune states. Either due to medications, or diseases like HIV and malignancies trigger seborrheic dermatitis.
Food. Sugar consumption, fast foods, Vit B including B12 malutilization, biotin deficiency, niacin deficiency, pyridoxine deficiency, zinc deficiency and excess alcohol consumption have all been found to increase the incidence of seborrheic dermatitis .
Drugs. Broad spectrum Antibiotics, OCP, Systemic Steroids, buspirone, chlorpromazine, cimetidine, ethionamide, griseofulvin, haloperidol, interferon alfa, lithium, methoxsalen, methyldopa, phenothiazines, psoralens, stanozolol are some of the medications that have been found to increase seborrheic dermatitis. The list is not complete.
Diseases. Seborrheic dermatitis may be the presenting feature of Parkinsonism and HIV infection. Incidence of seborrheic dermatitis is high in epilepsy, idiopathic post encephalitis, diabetes mellitus, paralytic states etc.
What are the Other Causes for Itchy Scalp?
Seborrheic dermatitis has to be differentiated from scalp dryness, contact allergy and psoriasis, all of which cause itchy and scaly scalp.
What are the Types of Seborrheic Dermatitis?
Infantile Seborrheic Dermatitis
Craddle cap
Trunk: Flexural, napkin (diaper) area
Leiner’s disease
Adult Seborrheic Dermatitis
Scalp: Dandruff and inflammatory types.
Face: Inflammatory and non inflammatory, with blepharitis(eye lid)
Trunk: Petaloid, pityriasiform, follicular, follicular, eczematous type
Generalized exfoliative erythroderma
What are the Complications of Seborrheic Dermatitis?
Psychosocial impact: Chronic visible disease may affect a person's self esteem.
Secondary bacterial infection with Staphylococcus aureus may occur, with typical impetigo with increased redness, oozing and crusting.
What is Leiner’s Disease?
Leiner's disease is a complication of seborrheic dermatitis in infants with C5 complement deficiency. There is usually a sudden confluence of lesions leading to a generalised scaling and redness of the skin. The child is severely ill with anemia, diarrhea and vomiting. Secondary bacterial infection is common.
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I have had seborrheic dermatitis for almost 10 years now. I went to a
hematologist "blood disorder doctor" and found out that I had
"folic acid deficiency anemia". I then began treatment of a
"prescription" of folic acid. Within a week, I could tell that
it was healing. That was over a year ago and I must say it has been quite
painful and debillitating spending hours when my head was going through
what I called a "breakout". The pain was so extreme that I
thought I was going to have to go to the hospital.
I now am
still healing VERY, VERY slowly and hopefully in the end will heal
completely.
I recommend the following to help alleviate the
pain and discomfort:
1) Paul Mitchell's Tea Tree Oil
Shampoo/Conditioner. Apply and leave it in.
2) AmLantin
Moisturizing Body Cream which does have 12% lactic acid which is an
alpha-hydroxy acid which helps to exfoliate the skin quicker. Expensive
but worth it.
3) Sarna Moisturizing Lotion with menthol. A
little pricey but again a nice refreshing relief of the itching.
4) Milk of Magnesia helps to dry up the irritated areas quicker but
makes a messy head so I put a bandana over my head.
I do
recommend if you think you have this condition, to "demand" to go
to a hematologist. I had to somewhat argue with my regular physician but
finally got an appointment and even though it has been EXTREMELY long and
painful, I can actually go out without a baseball cap on most days.
Sylvia Hart Lakeland, FL
Sep 24, 2009 9:45 PM
Guest :
My derm gave me a jar of 10% glycolic acid cream, to even out the
discoloration that SD leaves behind when it goes into remission (I'm not
Caucasian).
I have SD on my face, chin, neck, occasionally
chest. It's most bothersome on my face, chin and neck.
The
regimen he has me on---cleaning morning and night with Cerave LOTION (not
the cleaner), using Protopic ointment twice daily, and Clenia cream at
night only---has given me my face back.
He also has me using
Cerave cream to moisturize my face when needed.
My face is so
dry and sensitive that he has me using only a wet washcloth to exfoliate
while washing with the Cerave lotion. I find this really helps with the
scales that build up on the SD areas and makes the face look way less
"rough" and way smoother.
My question is, how does the
glycolic acid 10% restore skin tone? Like, how does it work on the skin to
even out the color? I've never used any of the "acids" before. I
have to be really careful not to touch or unconsciously scratch my face
before applying it, because it BURNS. And if I'm having an active flare on
my face, I can't apply it all because it's AGONY. Thanks in advance.
Sep 25, 2009 12:00 AM
Hanish Babu :
Glycolic acid is the most popular among AHAs(Alpha Hydroxy Acids) used in
sun damaged and ageing skin.Its action is multifacted- peeling, reduced
melanin syntheisis, enhanced melanin dispersion and increased collagen and
ground substance synthesis in the dermis through the stimulation of
fibroblasts.The overall effects are evening out the skin tone and
rejuvenation of the skin texture.
These are not to be applied on
raw or inflammed skin.